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Treatment was well-tolerated, with Grade 3 or higher TRAEs occurring in 6.3% of patients (2/32), manifested as anemia (hematologic) and fatigue (constitutional)

Deurne: MIM

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York JE, Walsh GL, Lang FF, Putnam JB, McCutcheon IE, Swisher SG , Komaki R, Gokaslan ZL

Individual responses vary, and therapy is tailored to each patients health goals and clinical needs

In AKI, the injured proximal tubular cells promote inflammation and fibrosis and develop into CKD [24]