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Deficits in brain GSH concentrations are associated with increases in oxidative damage [59,60], impaired ability to quench and export toxins [57,61], neuroinflammation [62], and increased NO toxicity and neuronal cell death [52]

It can be concluded that the 2 nd activity controller influences both diphenolase and tyrosinase activity and acts as a substrate selector, but it is not able to inhibit monophenolase activity

Arch Biochem Biophysics

(23-7727), a Grant-in-Aid for JSPS Scientific Research (B) (20390077) to K.M

Malignant mouse-liver tumors resembling human hepatoblastomas

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