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(1998) Early phagocytosis of glucose-6-phosphate dehydrogenase (G6PD)-deficient erythrocytes parasitized by Plasmodium falciparum may explain malaria protection in G6PD deficiency

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As a result of lipid peroxidation, HNE, secondary bioactive aldehyde, is produced at a high levels in several brain regions of late-stage AD subjects [89]