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Taken together, our data indicate that the loss of GSH triggered by Gclc deletion induces a defective mitochondrial accumulation in FoB, which may lead to compensation through upregulation of glycolytic metabolism

Besides, the serum biochemistry analysis and complete blood panel analysis (Supplementary Fig

10.1038/sj.emboj.7600543 EMBO J

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Pediatr Res 19:459466 Frye RE, Melnyk S, Macfabe DF (2013) Unique acyl-carnitine profiles are potential biomarkers for acquired mitochondrial disease in autism spectrum disorder

Medicare Advantage plans must cover B12 injections to at least the same extent as Original Medicare