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Supporting these mechanisms through peptides may not cure neurodegenerative conditions, but the research increasingly suggests they could significantly alter the trajectory of decline

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TB-500 is a synthetic fragment of Thymosin Beta-4 whose core proposed action is the sequestering of G-actin to regulate actin polymerization the process behind cell migration in wound healing and which has been linked preclinically to angiogenesis and reduced inflammation [3] [4]