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Notably, these alterations occur prior to the onset of inflammation and induce the upregulation of epithelial-derived cytokines such as thymic stromal lymphopoietin (TSLP) and interleukin-33 (IL-33), which subsequently activate the Th2 immune axis in a sustained manner (Jo et al., 2018

Antioxidant enzymes support survival of breast tumor cells deprived of ECM, implying that eliminating antioxidant enzyme activity in ECM-detached tumor cells may be an efficacious strategy to stop metastatic spreading (262)

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Future Perspectives and Emerging Directions Although CQDs have demonstrated broad biological applicability and favourable biocompatibility in conventional in vitro and animal studies, these models do not fully capture the complexity of human physiology [86]