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Depletion of mitochondrial methionine adenosyltransferase 1 triggers mitochondrial dysfunction in alcohol-associated liver disease

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This origin is what places MOTS-c in a separate research category from hGH-fragment or GHRH-analog compounds, since its activity is rooted in mitochondrial biology rather than the pituitary-hormone axis

BPC-157 does not bind a single canonical receptor

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