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Zhang P et al (2019) Senolytic therapy alleviates A-associated oligodendrocyte progenitor cell senescence and cognitive deficits in an Alzheimers disease model
Unlike healthy cells, tumor cells exhibit notably altered metabolic patterns to obtain increased energy and resources for cell proliferation (9,10)
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A variety of cellular stresses, including DNA damage can cause the induction of cellular senescence, which represents a multifaceted and highly heterogeneous cellular state characterized by irreversible growth arrest and importantly the secretion of a multitude of factors, including proteases and chemokines 2
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[258] [259] [261] The field that studies these interactions is known as pharmacomicrobiomics
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