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10.36685/phi.v6i1.327 419

Moreover, analysis of mass isotopomer contribution of U- 13 C glutamine to -GC and GSH confirmed the reduction in M + 5-labeled [ 13 C]--GC and M + 5-labeled [ 13 C]-GSH in the shACTL6A-expressing cells

Li, C., Chen, Y

Via mitochondrial malfunction and ROS activation caused by bifurcating metabolic pathways (reverse electron transport and lipid metabolism), nicotinamide therapy promotes cancer cell death in TNBC in humans and mice (115) ( Table 2 )