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Numerous investigations have concentrated on the epigenetic regulatory mechanisms involved in ferroptosis, and the epigenetic regulators in ferroptosis include histone modifications, lymphocyte-specific helicase, long non-coding RNA, deubiquitinase, and selenium [46]

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In vitro expansion of chondrocytes, a necessary step in ACI, results in the generation of senescent cells that adversely affect the quality and quantity of newly formed cartilage