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Wound Healing and Regeneration: FOXO4-DRIs senolytic action reduces senescent cell burden in chronic wounds, improving healing outcomes and preventing excessive fibrosis
While complement deposition is still understood as a central mechanism, it was recently demonstrated that induction of optic neuritis by human AQP4ab in mice is dependent on Interferon type I signaling (94)
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Based on previous work, the team knew that glutathione contributes to maintaining the tightly regulated, Goldilocks-like environment of the ER, where secretory and membrane proteins manufactured by ribosomes are folded for export
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