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In healthy mitochondria, SIRT3 interacts with ATP5O, while the low pH owing to the loss of membrane potential weakens the binding affinity between SIRT3 and ATP5O, leading to the redistribution of SIRT3 to other mitochondria substrates

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GCP guidelines emphasize that the safety of the patient is a primary concern, and only individuals who can safely participate should be included in clinical trials

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Despite these advances, several critical challenges continue to impede the practical clinical translation of CQDs [103]