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ROS also affect the activity and gene expression of sleep-regulatory neuronal populations, including orexinergic neurons and the SCN, where redox-sensitive transcriptional feedback loops modulate circadian timing (Wilking et al., 2012

If effects persist strongly, longer rest periods optimize cost efficiency without sacrificing results

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ARDS metabolic fingerprints: characterization, benchmarking, and potential mechanistic interpretation

Thus, less microglia recruitment in BPC 157 rats is important for the maintained neuroprotection, since microglia become activated as a response to neuronal damage, changing into an ameboid form that can migrate to sites of injury where they phagocytose debris and dying cells [65,66]