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The traditional view posits that microglia activated around senile plaques accelerate disease progression by releasing ROS, neurotoxins, and pro-inflammatory cytokines, thereby directly linking A deposition to neurodegeneration (145, 146)

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However, its benefits extend beyond its antioxidant properties

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Cancer cells have higher levels of ROS than normal cells, which promotes tumorigenesis, however, too much intracellular ROS triggers cell death and inhibits tumor progression