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Chondrocyte dedifferentiation was majorly characterized as the reduced expression of COL2 and ACAN and enhanced expression of type I collagen and versican (Charlier et al., 2019)

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Once injected subcutaneously, semaglutide binds to GLP-1 receptors in the hypothalamus (reducing appetite centrally), the stomach (slowing gastric emptying and prolonging satiety), and pancreatic beta cells (enhancing glucose-dependent insulin secretion)

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Followed by binding it to c-Met from tumor cells, a signaling pathway is formed, which leads to proliferation, metastasis, and angiogenesis, for example in brain, gastric, and head and neck cancers [2, 3]