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As a result, the main melanogenesis regulator MITF is elevated after UVR, followed by an increase in other melanosomal proteins such as TYR, TRP-1 and TRP-2, leading to the stimulation of melanogenesis [47]

The binding of ammonium to TNF- activates its death domain, recruits TRADD (Tumor Necrosis factor receptor-associated death domain protein) and FADD (Fas-associating protein with a novel death domain) proteins, forms a complex, activates Caspase-8, and subsequently Caspase-3, leading to apoptosis (147)