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Repair proceeds through overlapping phases: hemostasis, inflammation, proliferation (angiogenesis, fibroplasia, matrix deposition), and remodeling (maturation and cross-linking of the matrix)

The specific bioactivation mechanisms can vary depending on the drug and the enzyme(s) involved

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A more general mechanism to explain oxidative stress generated by electrochemically silent metals is based on the formation of glutathione conjugates and on their decomposition under physiological conditions to yield two toxic agents: the nano-particulate metal sulphides that deposit in organ parenchyma and the electrophilic dehydroalanine analogue of glutathione