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Mechanism of salinity tolerance in plants: physiological, biochemical, and molecular characterization
Binding induces autophosphorylation of the intracellular kinase domain on tyrosines 1131/1135/1136, recruits substrates IRS-1, IRS-2 and Shc, and activates two major signaling pathways: the PI3K-AKT-mTOR pathway (protein anabolism, cell growth, apoptosis inhibition) and the Ras-Raf-MEK-ERK pathway (cell proliferation, growth gene transcription)
Of note, these patients are also at high risk for cardiovascular disease
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We now propose a feasible mechanism: Denley and colleagues [119] have beautifully designed a study where they demonstrate how IR has a splice variant lacking exon 11 which confers the receptor affinity for IGF-1 and IGF-2
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