p53 and ribosome biogenesis stress: The essentials
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Key Points A large body of literature strongly suggests that cigarette smoke unfavourably influences all major stages of atherosclerosis as well as pathological atherothrombus formation Cigarette smoke contains oxidant compounds that cause oxidative stress in the endothelium, leading to endothelial dysfunction and injury, initiation of the atherosclerotic process, and subsequent formation of atherosclerotic plaques Cigarette smoke promotes the development of vulnerable plaques and plaque rupture by enhancing inflammation and activating matrix metalloproteinases Cigarette smoke causes platelet activation, and promotes platelet aggregation and platelet adhesion to sites of endothelial injury Exposure to cigarette smoke shifts the balance of haemostasis towards thrombus formation by enhancing blood clotting and, at the same time, reducing the fibrinolytic capacity This is a preview of subscription content, access via your institution Access options Subscribe to this journal Receive 12 print issues and online access $189.00 per year only $15.75 per issue Buy this article Purchase on SpringerLink Instant access to the full article PDF
However, if the smell does not disappear after 24 hours or is accompanied by smoke, your furnace could have a more serious problem
Brand: Premier Type: Cigarette filter tips for roll-your-own Count:
There is a wealth of health and personal benefits available to those who successfully stop and help can be sought through the full range of Smokefree support, which includes face-to-face advice, Smokefree app, Quit Kit, plus email and text programmes
