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Given the similarities in the biological mechanisms of both types of pollutants, it is plausible that the widespread exposure to a combination of ambient particle matter (PM10) and cigarette smoke may impact the level of E2 in women residing in an environment with higher levels of pollutants

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For nicotine studies, they always used sucrose as the positive controlthe substance they knew would activate reward circuits

But I think that, over the past couple of decades, it's come to stand for a big mass of other things too