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In the present study, we hypothesized that M-CS could induce more severe lung inflammation than Non-M-CS could via the additional action of menthol on TRPM8

Notably, Nrf2/ mice showed more obvious airway barrier dysfunction characterized by the abnormal expressions of E-cadherin, ZO-1, Bax and Bcl-2 under CS exposure

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In a transplantable model of BC the dual CCR1/CCR5 antagonist, Met-CCL5, impeded tumor growth and inhibits the migration of macrophages and lymphocytes into 410.4 tumors, which potentially implicated CCL5 in the facilitation of tumor-promoting macrophage or lymphocyte infiltration

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