Although the cytotoxicity in endothelial cells exposed to traditional cigarette smoke was greater, devices with higher heating temperatures exhibited an increased particulate phase of smoke, characterized by a higher presence of cytotoxic carbonyl compounds, resulting in reduced mitochondrial metabolic activity and eNOS activity in endothelial cells [31]
Normally, tiny hair-like structures in the airways (called cilia ) help sweep harmful material out of the lungs
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