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doi:10.3766/jaaa.17140

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Ongoing clinical and mechanistic studies are expected to expand use of GLP-1 receptor agonists, glucose-dependent insulinotropic polypeptide agonists and other metabolic hormones for the treatment of chronic kidney disease

These effects occur despite BPC-157 not directly increasing VEGF-A expression, suggesting the peptide works primarily through receptor sensitization rather than ligand upregulation

These include individuals with nutrient deficiencies or those seeking added metabolic support during structured weight loss plans: Patients with B12 deficiency who may experience fatigue and metabolic slowdown

Maria Guadalupe Cardenas, MD (NPI #1164426749, Texas MD License #J2933), a board-certified internist with over 40 years of experience