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Interestingly, while these age-causal CpGs were initially identified in blood [68], we observe the enrichment of age-causal CpGs in smoking-DMPs from the lung

found that differential DNA methylation of the muscarinic acetylcholine receptor 1 and 3 pathways was enriched in response to pesticide exposure

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Recent drug development efforts have led to the discovery of a highly potent bioengineered peptibody inhibiting Kir3.1/Kir3.4

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