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Recent research has found that mutations in the TCA cycle enzymes SDH, FH, and IDH resulted in a dysfunction of the TCA cycle and defects in mitochondrial metabolism in a wide range of human cancers [7, 8]

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In the presence of specific ferroptosis-inducing agents, baicalein exhibited superior anti-ferroptosis activity compared to established ferroptosis inhibitors such as ferrostatin-1, liproxstatin-1, desferrioxamine mesylate, and -mercaptoethanol (71, 74)

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