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No, it doesnt make sense

Protein-induced by vitamin K absence or antagonist-II (PIVKA-II), also known as des--carboxyprothrombin (DCP), is a prothrombin precursor that would normally be converted to native prothrombin via the action of a carboxylase that is vitamin-K dependent

While the underlying molecular basis is rather heterogeneous, potentially contributing to the suboptimal success rate of available therapeutic interventions, a number of common elements have been identified

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For example, by reducing oxidative stress levels, DNA oxidative damage, and modulating epigenetics, some nutrients and phytochemicals could have antioxidant/anti-inflammatory properties that can affect or prevent LC pathogenesis [167,168]