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T4SS and its effector molecule CagA are recognized as key virulence factors responsible for gastric cancer development

Personalized protocols based on genetics, oxidative stress biomarkers and metabolomic profiles will probably surface, allowing clinicians to tailor peptides to the patients unique oxidative and inflammatory fingerprint

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The treatment involves daily or frequent visits for infusions

These findings underscore the critical role of mitochondrial dysfunction in the pathogenesis of GWS and suggest that targeting impaired energy metabolism may offer novel therapeutic avenues