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Conversely, Mycobacterium tuberculosis ( M.tb ) infection stabilizes mitochondrial networks by inhibiting DRP1 activation, thus preventing premature host cell apoptosis and ensuring a replicative niche within macrophages (Xu et al., 2014)

Until well-designed clinical trials are conducted, BPC-157 should be considered investigational, and its use approached with caution

TB-500 is more connected with thymosin beta-4 style research, cell migration, and tissue remodelling

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The next question was how far the cross-reactive effect extended

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