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Tregs can also directly inhibit natural killer (NK) cell effector functions by the down-regulation of NKG2D receptors on the surface of NK cells caused by membrane-bound TGF- on Tregs (231)

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Longitudinal in vivo imaging of MAM dynamics in AP is lacking, making temporal relationships between ER stress, MAM remodeling, and cell death largely inferential

Of particular interest is the finding that GSH co-localizes to the nucleus at the onset of proliferation, which through redox changes can affect the activity of many nuclear proteins including histones [14,31]

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