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Xanthine oxidase and mitochondria contribute to vascular superoxide anion generation in DOCA-salt hypertensive rats

Upon SA accumulation the cellular redox potential becomes more negative by increasing GSH contents, which results in the reduction of NPR1 oligomers to a monomeric form

The observed downregulation of muscle atrophy-related ubiquitin ligases (Atrogin-1 and MuRF-1) is consistent with the reported capacity of SCFAs to modulate proteolytic pathways 56 , thereby potentially contributing to the amelioration of muscle metabolic dysfunction

doi: 10.1038/nature10350 60 ProvencherS.MaiV.BonnetS

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