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The positive role of glutathione in preventing hypertension may also be explained by the observations that high glutathione levels prevent platelet-derived growth factor (PDGF)-mediated production of ROS by NADH/NADPH oxidase, which is considered to be the main source of vascular ROS (7), (22)

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Besides, we demonstrated the shared mechanistic connections between chronic ethanol exposure and AD: oxidative stress and neuroinflammation, proved that chronic ethanol exposure may accelerate AD pathology by increasing A aggregation in mice and causing oxidative stress and neuroinflammatory impairments

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