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These findings suggest that trained immunity contributes to neuroinflammation and neurodegeneration in AD

The activation of ASK1, induced by H 2 O 2 or hypoxia/reoxygenation, leads to elevated levels of soluble receptor for VEGF (sFlt-1) [203], which has anti-angiogenic properties [150, 204]

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DNA damage triggers signaling molecules, resulting in increased production of proinflammatory chemicals and suppression of eNOS activity [112,113]

Xenobiotic-induced transcriptional regulation of xenobiotic metabolizing enzymes of the cytochrome P450 superfamily in human extrahepatic tissues

Inflammation, epigenetics, and metabolism converge to cell senescence and ageing: the regulation and intervention