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Despite the difficulties to quantify the ROS because of their labile nature, the stable end products of the reactive pathways may be used as reliable markers of oxidative stress in patients with asthma [7]

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JHU-083 has been demonstrated to be highly effective against EGFR-driven lung tumorigenesis, with the capacity to promote adaptive T-cell-mediated tumor-specific immune responses that enhance evasion [103]

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