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On the other hand, the cervical TME is frequently dominated by chronic inflammation and profound immunosuppression, creating a niche where malignant cells evade immune surveillance and thrive despite therapy [46,47,48]

Experiments were designed to determine the potential protective ability of NACA against different oxidative drugs

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By altering fluidity and permeability, lipid peroxidation compromises the cellular membrane and significantly disrupts overall cellular function [48,50]