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These somatic and signaling abnormalities are accompanied by increased oxidative stress associated with mitochondrial dysfunction both in ASD and AD [353]

Dilokthornsakul, W., Dhippayom, T., & Dilokthornsakul, P

In summary, these data suggest that glutathione plays a vital role in the detoxification of BH3 mimetics and that perturbation of GSH metabolism increases the sensitivity of AML cells towards BH3 mimetics