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Systematic reviews have also highlighted similar neuroinflammatory evidence in ME/CFS patients, though significant individual variability exists, with some studies failing to detect notable differences in TSPO binding (155)

These conditions may have soft tissue components that respond to treatment, but the underlying structural issues persist

The synergy of amyloid 1-42 and oxidative stress in the development of Alzheimers disease-like neurodegeneration of hippocampal cells

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